Lavender Flower is a flowering plant in the mint family with documented use for over 2,500 years, extending back to ancient Egypt.
✓ Healthy aging
✓ Promote calm
✓ Improve sleep quality
✓ Support relaxation
How it works:
Used in cooking, essential oils, and perfumes, recent research has shown beneficial results used both orally and topically to promote healthy aging of skin and body through interactions with prostanoids, proinflammatory cytokines, and histamine pathways. Clinical research supports the use of lavender to promote natural, healthy stress response, sleep quality, and relaxation.
Safe and Effective Dosage:
Dosage is highly varied due to its use in concentrated oil, teas, and foods, with dosage ranging from 80 mg to over 1000 mg.
RESEARCH &CLINICAL STUDIES
"Lavender and the Nervous System"
Lavender is traditionally alleged to have a variety of therapeutic and curative properties, ranging from inducing relaxation to treating parasitic infections, burns, insect bites, and spasm.
There is growing evidence suggesting that lavender oil may be an effective medicament in treatment of several neurological disorders.
Several animal and human investigations suggest anxiolytic, mood stabilizer, sedative, analgesic, and anticonvulsive and neuroprotective properties for lavender.
These studies raised the possibility of revival of lavender therapeutic efficacy in neurological disorders.
In this paper, a survey on current experimental and clinical state of knowledge about the effect of lavender on the nervous system is given.
"Effect of Lavender (Lavandula angustifolia) Essential Oil on Acute Inflammatory Response"
Lavandula angustifolia is a plant of Lamiaceae family, with many therapeutic properties and biological activities, such as anticonvulsant, anxiolytic, antioxidant, anti-inflammatory, and antimicrobial activities.
The aim of this study was to evaluate the effect of Lavandula angustifolia Mill. essential oil (LEO) on acute inflammatory response. LEO was analyzed using gas chromatography-mass spectrometry (GC-MS) and nuclear magnetic resonance spectroscopy (NMR) methods and showed predominance of 1,8-cineole (39.83%), borneol (22.63%), and camphor (22.12%).
LEO at concentrations of 0.5, 1, 3, and 10 μg/ml did not present in vitro cytotoxicity. Additionally, LEO did not stimulate the leukocyte chemotaxis in vitro. The LEO topical application at concentrations of 0.25, 0.5, and 1 mg/ear reduced edema formation, myeloperoxidase (MPO) activity, and nitric oxide (NO) production in croton oil-induced ear edema model.
In carrageenan-induced paw edema model, LEO treatment at doses of 75, 100, and 250 mg/kg reduced edema formation, MPO activity, and NO production. In dextran-induced paw edema model, LEO at doses of 75 and 100 mg/kg reduced paw edema and MPO activity.
In conclusion, LEO presented anti-inflammatory activity, and the mechanism proposed of LEO seems to be, at least in part, involving the participation of prostanoids, NO, proinflammatory cytokines, and histamine.